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Recently, evidence has demonstrated that induction of BDNF expression via stimulating/modulating activity of neurotransmitter receptors, and application of small molecules that activate TrkB are beneficial strategy for both neuroprotection and neuroregeneration

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For instance, this study (146) utilized a neonatal maternal separation stress model in mice to simulate irritable bowel syndrome (IBS) and identified neuronal nitric oxide synthase (nNOS) as a novel and reliable biomarker for interstitial cells of Cajal stimulation in IBS